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CaseDiscussionAxialarterialphaseAxialarterialphaseAxialvenousphaseCoronalarterialphaseSagittalvenousphaseCrohndiseaseAninflammatorydiseaseofthegastrointestinaltractthattypicallyhasanindolentcourseCharacterizedbyintestinalulceration,strictures,andfistulaformationCommonlyaffectsyoungadults,smallbowel,particularlytheterminalileumSmallbowelinvolvementinCrohndiseaseistypicallytransmural,withskiplesionsCTandMRICTandMRIUsefulfordifferentiatingbetweenactiveandfibroticbowelstricturesAllowingvisualizationoftheentirethicknessofthebowelwallDepictingextraentericinvolvementProvidingmoredetailedandcomprehensiveinformationabouttheextentandseverityCombsignFathalosignBowelwallenhancementBowelwallthicknessStrictureandfistulaMesenteric/intra-abdominalabscess(15%-20%)orphlegmonformationUlcerationsandlossofhaustrationCreepingfatCombsignProminenceofthevasarectaadjacenttotheinflamedloopofbowelTransmuralextensionofinflammationacrosstheserosaandtoengorgementofthehyperemicvasarectasurroundingtheinflamedbowelsegmentNotpathognomicofCrohndiseaseFathalosignInfiltrationofthesubmucosawithfat,betweenthemuscularisandthemucosaConfusedwiththefatringsignofmesentericpanniculitisNearlypathognomonicofinflammatoryboweldisease(Crohndiseaseandulcerativecolitis)BowelwallenhancementTheresultofincreasedvascularpermeabilityandangiogenesisThemostsensitiveindicatorofactiveCrohndiseaseEnhancementcanbegradedbycomparingtotheprecontrastimagesMinorincreasedenhancementModerateenhancementMarkedenhancementNoabnormalenhancement:equivalenttonormalbowelwallHomogeneousBowelwallthicknessNormalbowelwallthickness:lumendistended,1-2mm;lumencollapsed,3-4mmMild:3-5mmModerate:5-7mmMarked:>7mmOneofthemostcommonsigns,butnotspecificCorrelateswellwiththeseverityofthediseaseactivityMeasurewhenlumendistendedwellBlackborderartifactscandistortthicknessmeasurementsStrictureAcomplicationofCrohndiseaseReversiblestricturesproducedbyactivediseaseAlackofenhancementandlossofstratificationmightbeseeninthe